A tigroid fundus, also called a tessellated fundus, describes a striking striped or tiger-skin appearance visible on dilated fundus examination, and recognizing what actually produces this pattern, thinning of the retinal pigment epithelium unmasking the underlying choroidal blood vessels, explains both why it looks the way it does and why its presence carries real information about the health and risk profile of the eye it is found in.

Clinical eye photograph illustrating Tigroid Fundus

What Produces the Striped Appearance

The retinal pigment epithelium normally contains enough pigment to substantially obscure the underlying choroidal vasculature from view during fundus examination, giving the normal fundus its relatively uniform, orange-red appearance.

When the retinal pigment epithelium thins, as commonly occurs with axial elongation in myopia, this pigment layer becomes less effective at masking the choroid beneath it, and the large choroidal vessels become directly visible as alternating light and dark stripes, producing the characteristic tigroid or tessellated pattern.


Myopia as the Dominant Cause

A tigroid fundus is most commonly seen in myopic eyes, and its prevalence and prominence generally increase with the degree of myopia and, to some extent, with age, reflecting the progressive retinal pigment epithelial thinning that accompanies axial elongation of the eye over time.

  • Mild to moderate tigroid change is common and often not, by itself, a marker of significant pathology
  • More pronounced tessellation, particularly in high myopia, is more often associated with the broader spectrum of myopic fundus changes and should prompt a more thorough assessment for other signs of pathologic myopia

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What a Tigroid Fundus Signals in High Myopia

In the context of high myopia, a prominent tigroid appearance is often one of the earliest visible fundus changes reflecting the underlying process of progressive chorioretinal thinning, and its presence should prompt a careful, systematic search for other, more directly vision-threatening features of pathologic myopia.

  • Posterior staphyloma, a structural outward bulging of the thinned sclera and overlying tissue (see posterior staphyloma)
  • Lacquer cracks, breaks in Bruch membrane that can develop within areas of significant chorioretinal thinning
  • Areas of more focal or diffuse chorioretinal atrophy, which can progress over time and, in advanced cases, threaten central vision if they extend into or near the macula
  • Increased risk of myopic choroidal neovascularization, related to the same underlying thinning and structural change

Clinical Evaluation

  • Dilated fundus examination characterizing the extent and distribution of the tessellated pattern, along with a systematic search for any coexisting signs of more advanced myopic degeneration
  • Measurement of axial length and refractive error, since the degree of myopia correlates with both the prominence of tessellation and the broader risk of associated pathologic changes
  • Wide-field fundus photography supports documentation and comparison over time, useful for monitoring any progression of associated chorioretinal changes
  • Optical coherence tomography can help assess retinal pigment epithelial and choroidal thickness directly, and screen for any early signs of the more serious complications associated with high myopia

Distinguishing a Simple Tigroid Fundus From Pathologic Myopia

The key clinical distinction is between a tigroid appearance as an isolated, relatively benign finding, common in mild to moderate myopia, and tessellation occurring as one component of a broader pattern of pathologic myopic degeneration, which carries meaningfully different implications for monitoring and long-term visual prognosis.

This distinction is made by assessing the fundus as a whole, specifically looking beyond the tessellation itself for the other structural changes, staphyloma, chorioretinal atrophy, and lacquer cracks, that together define more advanced myopic maculopathy.


Management

A tigroid fundus itself requires no treatment, since it is a descriptive finding reflecting underlying retinal pigment epithelial thinning rather than a disease process in its own right.

When it occurs in the context of high myopia, management is directed at appropriate monitoring for the associated complications of pathologic myopia, including regular dilated examination and patient education about symptoms, such as new distortion or a scotoma, that would warrant prompt reevaluation for a developing complication like choroidal neovascularization.


Prognosis

An isolated tigroid fundus in a patient with mild to moderate myopia carries an excellent prognosis and generally needs nothing beyond routine, age-appropriate eye care.

When tessellation is prominent and accompanies other signs of high, pathologic myopia, the prognosis is tied to that broader condition, and ongoing monitoring for progressive chorioretinal atrophy and neovascular complications becomes the central, appropriately more vigilant, focus of long-term care.


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References

  1. Ohno-Matsui K, Kawasaki R, Jonas JB, et al. International photographic classification and grading system for myopic maculopathy. Am J Ophthalmol. 2015;159:877-883.
  2. Hayashi K, Ohno-Matsui K, Shimada N, et al. Long-term pattern of progression of myopic maculopathy: a natural history study. Ophthalmology. 2010;117:1595-1611.
  3. Fang Y, Yokoi T, Nagaoka N, et al. Progression of myopic maculopathy during 18-year follow-up. Ophthalmology. 2018;125:863-877.
  4. Grossniklaus HE, Green WR. Pathologic findings in pathologic myopia. Retina. 1992;12:127-133.

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  1. What produces the striped appearance of a tigroid fundus?